Author

Zhu Zhu

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Open access Jul 2026

CCR5 drives glucose metabolic reprogramming to control microglial synaptic pruning via histone lactylation in a mouse model of depression.

A CCR5-driven immune-metabolic-transcriptional axis in microglia that underlies synaptic deficits in depressive-like behaviors is indicated, offering potential targets for therapeutic intervention.

Ying-Ying Jiao, Zhu Zhu, Rui-An Wang et al. · Cell Reports · 0 citations