Multi‐Omics Analysis Reveals That Aβ42‐ Me3 Co‐Aggregation Induces Mitophagy Blockade in Early Stages of Alzheimer's Disease
Mitochondrial dysfunction is one of the earliest pathological features of Alzheimer's disease (AD), preceding overt neurodegeneration and cognitive decline. Amyloid‐β (Aβ) accumulation has long been considered a central pathogenic event in AD, yet how Aβ toxicity is mechanistically linked to mitochondrial impairment du...