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A high-resolution cellular and temporal framework of SZ susceptibility is established, implicating mature associative IT microcircuits, deep-layer thalamocortical-regulating networks, and early developmental specification windows as primary points of genetic convergence in SZ.
Sleep disorders, inflammatory dysregulation, and anxiety symptoms constitute an interconnected network associated with SI in MDD, and the importance of comprehensive, integrated interventions is highlighted.
High BMI correlates with more severe depressive symptoms and increased inflammatory status in adolescents with MDD, especially among females, suggesting that interventions targeting immune and metabolic pathways may offer additional therapeutic benefits.
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