Author

Yi Xiao

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Sep 2026

Exosomal METTL16 from mesenchymal stem cells promotes malignant progression and M2 macrophage polarization in acute myeloid leukemia via IGF2BP2-dependent GPX1 m6A modification.

BACKGROUND Acute myeloid leukemia (AML) progression relies on complex crosstalk with the bone marrow microenvironment, particularly M2 macrophage polarization. Exosomes mediate intercellular communication, while N6-methyladenosine (m6A) modifications drive tumor malignancy. However, the role of exosomal m6A regulators...

Zhen Shang, Mi Zhou, Si-Jia Yan et al. · Biochimica et Biophysica Act... · 0 citations

Review Open access Jul 2026

Cellular Immunotherapy for Acute Myeloid Leukemia: From Bench to Bedside

Acute myeloid leukemia is a clonal malignant disease that arises from hematopoietic stem cells and is characterized by high genetic heterogeneity. AML has a poor prognosis, with a low 5-year survival rate, and is typically treated with chemotherapy. The management of AML, particularly for cases that are relapsed or r...

Yu Wang, Jiaying Wu, Xiaobing Huang et al. · Immunotherapy Advances · 0 citations