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Exposure to SO2 and specific EDCs (both individually and as a mixture) is significantly associated with localised PCa, and weighted quantile sum regression analysis demonstrated a significant positive association between the mixture of EDCs and PCa risk.
These findings defined the DPT+‐complement‐macrophage axis as an actionable vulnerability and position TSPAN1 and NRXN1 as therapeutic entry points to disrupt ADT‐driven tumor ecosystem remodeling in prostate cancer.
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