EFTUD2-Regulated Alternative Splicing of MSH5 Drives Radioresistance in Recurrent IDH-Mutant Glioma.
Therapeutic disruption of this splicing program may represent a strategy to enhance the radiation response in recurrent IDH-mutant glioma.
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Therapeutic disruption of this splicing program may represent a strategy to enhance the radiation response in recurrent IDH-mutant glioma.
A transcriptomic benchmark for current iMG models is provided and their use in investigating microglia-glioma interactions is supported, to provide a transcriptomic benchmark for current iMG models.
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