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Tz-Chuen Ju

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Open access Jul 2026

IL17A disrupts autophagy-lysosomal function and lysosome reformation through the GSK3B-TFE3 signaling pathway in Huntington disease.

Therapeutic neutralization of IL17A with a monoclonal antibody (IL17A mAb) ameliorates disease phenotypes in R6/2 HD mice, improving motor performance, extending survival, and reducing gliosis, and highlights IL17A inhibition as a promising therapeutic strategy for targeting autophagy-lysosomal dysfunction in HD.

Kai-Po Chen, Tz-Chuen Ju · Autophagy · 0 citations