Vision-language-action (VLA) models have become a dominant paradigm for generalist embodied agents, demonstrating strong complex and long-horizon task completion in structured settings. Yet it remains an open question whether current VLA systems can benefit from more effective architectural design, scale to substantially larger and more heterogeneous data regimes, and achieve broader generalization across tasks and embodiments. To this end, we present GigaBrain-0.7, an embodied foundation model with substantially improved generalization across diverse robot embodiments. Specifically, GigaBrain-0.7 unifies understanding, prediction, and action through a three-system architecture, scales pretraining to over 37,000 hours of heterogeneous embodied data, and introduces one-stage alignment training that jointly optimizes vision-language understanding and multi-embodiment action generation. Compared with the preceding GigaBrain-0 series and prior state-of-the-art models including $\pi_{0.5}$, GigaBrain-0.7 achieves substantial improvements in foundation zero-shot capabilities, language-conditioned instruction following, and post-training task success rates. In particular, on our in-house Maker H01 platform and mainstream robot embodiments, GigaBrain-0.7 demonstrates strong task adaptability and completion ability across both home and industrial scenarios. All training code and pretrained model weights will be released.
GigaBrain Team, Angen Ye, Axiang Sun et al.· 0 citations
Diabetic patients face heightened risks for cardiac implantable electronic devices (CIED) infections. Herein, we developed a near-infrared (NIR)-activated porphyrin polymer (PTCPP) for photodynamic therapy of CIED infections, which achieves a 100% antibacterial rate with a 22% increase in anti-biofilm efficacy compared to porphyrin (TCPP) in vitro. In diabetic rats, a 93% bactericidal rate is further demonstrated by PTCPP, along with a 97% reduction in the levels of pro-inflammatory factors. It is found that singlet oxygen (1O2) and hydroxyl radicals (•OH) are the main reactive oxygen species (ROS), which can eliminate methicillin-resistant Staphylococcus aureus by disrupting mature biofilms. The immunomodulatory effect of PTCPP stems from dynamic regulation of macrophage polarization. Under irradiation, ROS induce M1 activation to eliminate pathogens, followed by metabolic reprogramming that promotes healing-conducive M2 polarization, thereby accelerating tissue repair. This work showcases the great potential of synergistic photodynamic-immunomodulatory strategies for the next-generation prevention of complex implant-related infections.