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Prenatal fluoride exposure does not appear to affect pregnancy outcomes, but preliminary evidence suggests that higher fluoride levels may affect the risk of LGA depending on timing of exposure during pregnancy.
Prenatal serum PFAS concentrations were associated with alterations in maternal DNA methylation, which in turn were associated with infant birth size, which in turn was associated with infant birth size.
This is the first epidemiologic study to evaluate prenatal exposure to aromatic amines in relation to infant cognition and extends concerns about aromatic amines beyond carcinogenicity to early neurodevelopment, highlighting a previously unrecognized pathway of potential harm during a critical developmental window.
It is found that several AAs are associated with higher levels of oxidative stress, highlighting one potential mechanism linking AAs to adverse maternal-child health outcomes.
Global analysis of the maternal lipidome during early pregnancy revealed diverse bioactive lipid involvement in the PFAS-SGA association and if antioxidants and anti-inflammatory nutrients would attenuate exposure effects on adverse maternal–child health outcomes.
Overall, maternal dietary factors may influence susceptibility to PFAS-related fetal growth effects and may modify the inverse associations between prenatal PFAS exposure and birthweight for gestational age z-scores.
Prenatal OP exposure was not associated with autism-related traits in childhood and results were consistent across sex-stratified models, secondary analyses using quantile and logistic regressions, various sensitivity analyses, and after adjustments for dietary confounders.
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