Open access
Sep 2026
mtROS-NF-κB signaling supports ILC3 function and survival during sepsis-induced intestinal injury
Findings identify a mitochondria-associated ROS-NF-κB program that sustains protective cytokine production and survival in residual intestinal ILC3s during sepsis, although it is insufficient to prevent ongoing ILC3 loss and intestinal injury.
Li-Min Chen, Hao-Cheng Zhang, Si-Si Huang et al.
· Frontiers in Immunology · 0 citations