GALNT6-mediated O-glycosylation suppresses NLRP3 inflammasome activation to alleviate atopic dermatitis.
AIMS Atopic dermatitis (AD) is characterized by chronic cutaneous inflammation, but the endogenous mechanisms that restrain excessive inflammatory activation remain unclear. This study investigated the role of GALNT6 in AD and its potential association with NLRP3 inflammasome regulation. MATERIALS AND METHODS Public...