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It is suggested that mesenchymal progenitor cells do not commit to a myogenic fate during cancer cachexia, and non-myogenic progenitors might indeed be capable of adopting a myogenic fate during cancer cachexia.
An ex vivo platform that integrates microelectrode array stimulation with simultaneous recording and single-nucleus genomics from resected temporal cortex obtained from patients undergoing neurosurgery finds that stimulation strengthens cell assemblies and then linked this effect to cell-type-specific gene regulatory n...
Findings highlight a HuR-SerpinB9 axis that regulates T-cell senescence and persistence, offering a potential therapeutic target in cancer and autoimmune diseases.
It is demonstrated that CHAMP1 mutations disrupt multiple aspects of neuronal development, including homologous recombination (HR) dysfunction in NPCs, membrane excitability, ion channel function, and synaptic connectivity.
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