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Sponsored AI Networking Cookbook: Practical recipes for AI-assisted network automation and development AI Networking Cookbook: Practical recipes for AI-assisted network automation and development

Author

S. Kotlyarov

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Open access Sep 2026

γδ T-cells and the peripheral blood T-cell profile in pulmonary tuberculosis, chronic obstructive pulmonary disease, and their comorbidity

Introduction. The combination of chronic obstructive pulmonary disease (COPD) and pulmonary tuberculosis (TB) is a clinically significant comorbidity. The role of αβ- and γδ-T-cell immunity in their isolated and combined course remains insufficiently studied. Aim. To establish features of the subpopulation composi...

D. Oskin, S. Kotlyarov, E. N. Fedyaeva · 0 citations
Review Open access Sep 2026

Canonical and Non-Canonical Regulators of Lipid Metabolism in the Liver and the “Liver–Other Organs” Axes

Metabolic dysfunction-associated steatotic liver disease (MASLD) is the most common chronic liver disease and is closely associated with obesity, insulin resistance, and the recently conceptualized cardiovascular–kidney–metabolic syndrome. For decades, the pathogenesis of steatosis has been viewed primarily through the...

S. Kotlyarov, A. Kotlyarova, R. Catanzaro et al. · 0 citations
Review Open access Sep 2026

Ferroptosis in metabolic dysfunction-associated steatotic liver disease

Metabolic dysfunction-associated steatotic liver disease (MASLD) is a widespread chronic liver disease that can progress from steatosis to steatohepatitis, fibrosis, cirrhosis, and hepatocellular carcinoma. Its pathogenesis involves lipotoxicity, iron metabolism disorders, and oxidative stress—the same three processes...

S. Kotlyarov, Aleksandra Iskrina, A. Kotlyarova · 0 citations
Review Open access Sep 2026

Features of Lipid Disorders in Cardiovascular–Kidney–Metabolic Syndrome

Cardiovascular–kidney–metabolic (CKM) syndrome is driven by the interplay of visceral adiposity, insulin resistance, chronic inflammation, oxidative stress, and progressive kidney dysfunction. These mechanisms generate a distinct atherogenic dyslipidaemic phenotype characterised by elevated triglycerides (TG), reduced...

A. Ceasovschih, Y. Şener, M. Arıcı et al. · 0 citations

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