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This work shows that high-frequency stimulation alters the subsequent properties of neurotransmitter release to confer a presynaptic form of homeostasis, independent of postsynaptic receptors, and shows that the activity-dependent regulation of release requires the t-SNARE syntaxin.
Accumulation of wild-type α-synuclein in dopamine neurons of the Substantia Nigra pars compacta (SNc) is a pathological hallmark of Parkinson’s disease (PD), which commonly precedes neurodegeneration. Yet how α-synuclein accumulation affects nigrostriatal neurotransmission early in the progression of PD remains poorly...
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