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R. Kaundal

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Aug 2026

4-Octyl itaconate attenuates cerebral ischemia reperfusion injury by mitigating mitochondrial dysfunction mediated neuronal apoptosis and ferroptosis via the Nrf2/HO-1/SLC7A11/GPX4 axis.

Ischemic stroke is an unmet therapeutic need, characterised by excessive oxidative stress, mitochondrial impairment, blood-brain barrier (BBB) damage, and neuronal cell death. 4-Octyl itaconate (4-OI) is known for its promising antioxidant properties; however, its effects in cerebral ischemia/reperfusion (I/R) injury h...

Chandan Chauhan, Ravinder K. Kaundal · 0 citations

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