Aug 2026
NOV promotes renal ischemia-reperfusion injury by driving TFRC-dependent ferroptosis.
Findings identify the NOV-TFRC axis as a regulator of ferroptotic tubular injury and suggest NOV as a potential therapeutic target in I/R-AKI.
Yan-Lu Xin, Xiao Sun, Ningxin Zhang et al.
· Cellular Signalling · 0 citations