Review
Open access
Oct 2026
Endoplasmic reticulum–mitochondria contact sites in benign prostatic hyperplasia: evidence integration and mechanistic hypotheses
Evidence from BPH studies, including GRP78 upregulation, mitochondrial reactive oxygen species (ROS) accumulation, NLRP3 inflammasome activation, DRP1-associated abnormalities in mitochondrial dynamics, and resistance to apoptosis are integrated.
Lei Liu, Min-Hao Fu, Zhen-Ting Wang
· Frontiers in Immunology · 0 citations