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Author

Michael Y. Mi

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3 papers indexed here

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Open access Oct 2026

Proteomic signatures reveal why not all apolipoprotein B-containing lipoproteins are equal in atherosclerosis

All apolipoprotein B (apoB)-containing lipoproteins are causal risk factors for coronary artery disease (CAD). However, triglyceride-rich lipoproteins (TRL) and lipoprotein(a) [Lp(a)] exhibit greater per-particle atherogenicity than low-density lipoprotein (LDL) through mechanisms that remain incompletely underst...

J. Morze, E. Bjornson, Michael Y. Mi et al. · 0 citations
Open access Aug 2026

Genetic drivers of protein changes over time: Findings, considerations, and approaches in TOPMed cohorts and UK Biobank

Age is a major risk factor for many diseases, but the biological processes driving aging are heterogeneous across individuals. Efforts to untangle differences between chronological and biological age have focused on identifying age-associated markers, such as ’omics clocks. Many ’omics features, including proteins, are...

M. Gillman, H. Chen, A. Howard et al. · 0 citations
Open access Aug 2026

Apolipoprotein B-containing lipoproteins: distinct roles in atherosclerosis revealed by plasma proteomic profiling.

Beyond arterial lipid deposition, TRL and Lp(a) converge on inflammatory and vascular remodeling pathways that, under the assumptions of observational mediation analysis, may explain their excess per-particle atherogenicity compared with LDL.

J. Morze, E. Björnson, Michael Y. Mi et al. · 0 citations

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