LILRB4 Attenuates Ischemic Brain Injury by Inhibiting Microglial Necroptosis via a RIPK3‐Dependent Mechanism
Microglia are central mediators of neuroinflammation following ischemic stroke. Our previous multi‐omics data revealed significant upregulation of leukocyte immunoglobulin‐like receptor subfamily B member 4 (LILRB4) in microglia after transient middle cerebral artery occlusion (tMCAO), but its functional role remains u...