Modulatory effects of S1QEL on OxLDL-induced metabolic alterations and inflammatory responses in human monocytes
S1QEL effectively suppresses both glycolytic reprogramming and pro-inflammatory cytokine expression induced by OxLDL in human monocytes, underscore the role of mitochondrial complex I-derived ROS in monocyte activation and highlight S1QEL as a potential therapeutic agent for targeting inflammation in aging and several...