Author

Li-Di Zhang

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Open access Sep 2026

mtROS-NF-κB signaling supports ILC3 function and survival during sepsis-induced intestinal injury

Findings identify a mitochondria-associated ROS-NF-κB program that sustains protective cytokine production and survival in residual intestinal ILC3s during sepsis, although it is insufficient to prevent ongoing ILC3 loss and intestinal injury.

Li-Min Chen, Hao-Cheng Zhang, Si-Si Huang et al. · Frontiers in Immunology · 0 citations