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Jun-Na Jiao

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Open access Sep 2026

Activation-induced cytidine deaminase sustains glycolytic flux through MYST1 acetyltransferase-dependent lactylation in hepatocellular carcinoma.

Hepatocellular carcinoma (HCC) exhibits profound metabolic reprogramming, yet the mechanisms that sustain persistent glycolytic flux remain poorly defined. In this study, we identify activation-induced cytidine deaminase (AID) as a non-canonical metabolic regulator that drives HCC progression through a self-reinforcing...

Jun-Na Jiao, Kai Zhang, Bai-Ge Li et al. · 0 citations

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