A novel LncRNA TUG1/miR-30d/e-3p/SNAI2 ceRNA axis drives dermal fibrosis in systemic sclerosis via direct transcriptional activation of COL1A1.
Findings identify the TUG1-miR-30d-3p/miR-30e-3p/miR-30e-3p-SNAI2-COL1A1 axis as a potential therapeutic target in SSc skin fibrosis.