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Author

Ji-Fu Zheng

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Open access Jul 2026

HMGB1 ubiquitination inhibition and extracellular secretion, mediated by m6A modified-exosomal lncRNA MALAT1, creates a communication between NK92-MI cells and tumor cells to aggravate acute lymphoblastic leukemia.

All cells-derived exosomal MALAT1 was up-regulated by METTL14-mediated m6A modification, and subsequently restrained HMGB1 ubiquitination and degradation in NK92-MI cells, which resulted in adriamycin resistance and malignant growth of ALL cells.

Xiaofang Hong, Daiyan Yang, Jianhao Xing et al. · 0 citations

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