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This work identifies a novel molecular mechanism for TDP-43 dysfunction, opening novel avenues for understanding disease pathogenesis and developing much needed pathology biomarkers.
An integrated wet-lab and computational platform designed to overcome barriers to population-scale long-read RNA sequencing, providing a scalable, community-ready framework for isoform-resolved transcriptomics in neurodegeneration, aging, and complex brain disease.
Exposure of T1D-associated cytokines to primary human ductal cells altered cargo of secreted EVs, suggesting a previously unknown ductal cell communication pathway with a potential implication for modulating beta cells during T1D progression.
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