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[This corrects the article DOI: 10.3389/fnagi.2026.1849634.].
This review dissects the hierarchical organization of the synaptome, arguing that synaptic decay is not a generic process of attrition but a specific collapse of subsynaptic domains (SSDs) and trans-synaptic nanocolumns, considered here within the framework of the tetrapartite synapse.
It is suggested that glial lncRNAs are crucial regulators of neuroinflammation in cognitive disorders, and their ability to modulate pathways such as the NLRP3 inflammasome makes them promising diagnostic biomarkers and therapeutic targets.
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