Abstract Background Evidence of how parenting stress evolves during early childhood is limited. We aimed to identify parenting stress trajectories involving children aged 1 month to 3 years, and to determine their perinatal predictors. Methods Data were obtained from 3133 mothers in the Tokyo Teen Cohort (TTC), a population‐based cohort of children born between 2002 and 2004. Parenting stress was self‐recorded in the Maternal and Child Health Handbook (MCHH) at eight time points between 1 and 36 months postpartum. Latent class growth analysis identified trajectories; multivariate multinomial logistic regression analyzed baseline predictors from the MCHH or the TTC first wave. Results Five distinct trajectories were identified: consistently low (n = 1,691, 54.0%), consistently high (n = 158, 5.0%), consistently moderately high (n = 719, 22.9%), high‐decreasing (n = 299, 9.5%), and low‐increasing (n = 266, 8.5%). Compared with the consistently low group, primiparity predicted all trajectories with initially high stress: consistently high (adjusted odds ratio [aOR] 1.64, 95% confidence interval [CI] 1.13–2.37), consistently moderately high (aOR 1.92, 95% CI 1.57–2.35), and high‐decreasing (aOR 1.87, 95% CI 1.38–2.54). Preterm birth also predicted the consistently high trajectory (aOR 2.38, 95% CI 1.31–4.32). Maternal alcohol consumption during pregnancy (aOR 1.33, 95% CI 1.06–1.67) and male sex (aOR 0.80, 95% CI 0.66–0.97) predicted the consistently moderately high trajectory. High maternal education (aOR 1.58, 95% CI 0.99–2.52) tended to predict the low‐increasing trajectory. Maternal age, chronic health conditions, smoking during pregnancy, single‐parent household, and history of infertility treatment were not significant predictors. Conclusion Parenting stress follows heterogenous trajectories, necessitating repeated assessments during the first 3 years. Integrating systematic, self‐recorded screenings into routine check‐ups could facilitate the timely identification of evolving stress patterns. Recognizing early predictors, including primiparity and preterm birth, can further assist in identifying mothers warranting closer longitudinal monitoring.
Nao Oikawa, Mariko Hosozawa, S. Yamasaki et al.· JCPP Advances· 0 citations
AIMS
Non-fasting triglyceride measurements have become increasingly common in screening for atherosclerotic cardiovascular disease. However, evidence of its predictive value for diabetes mellitus is limited to only a few studies.
METHODS
This prospective cohort study included two groups of individuals aged 40-74 years without diabetes mellitus: 7,673 who provided non-fasting blood samples and 4,055 who provided fasting samples. We used annual health checkup data to identify new cases of diabetes mellitus, employed Cox proportional hazard models to estimate hazard ratios, and conducted decision curve analyses to assess clinical utility.
RESULTS
During a median follow-up of 13 years, 727 and 444 participants were diagnosed with diabetes mellitus in the non-fasting and fasting groups, respectively. After adjusting for the glucose levels and other potential confounders measured at baseline, both non-fasting and fasting triglyceride levels were positively associated with the hazard of diabetes mellitus; the hazard ratios (95% confidence intervals) for the highest versus lowest quartiles were 1.99 (1.50-2.64) and 1.58 (1.11-2.26), respectively. Adding non-fasting or fasting triglyceride levels to a model based on traditional predictors can slightly improve the net benefit for 15-year risk prediction, corresponding to an approximate gain of 1 to 2 additional individuals per 1,000 screened. The improvement was slightly greater under non-fasting conditions than under fasting conditions.
CONCLUSION
Both the non-fasting and fasting triglyceride levels were associated with an increased hazard of diabetes mellitus. Adding the triglyceride levels can therefore provide modest but potentially useful supplementary information for diabetes prediction, particularly under non-fasting conditions.
Jia-Qi Li, H. Imano, K. Yamagishi et al.· Journal of atherosclerosis a...· 0 citations
We use cookies to run the site and, with your consent, for analytics and to show ads.
See our Cookie Policy.