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Genetic and Immunologic Interactions Driving STAT3 Signaling and Macrophage Polarization in Acute Myeloid Leukemia
Łukasz Szymański, AL Deng, K. O. Ahmed et al.
· 0 citations
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Open access
Aug 2026
Targeted mutational profiling of Fms-like tyrosine kinase 3, nucleophosmin 1, and isocitrate dehydrogenase 2 in acute myeloid leukemia: Associations with remission, subtypes, and survival
Acute myeloid leukemia is a genetically heterogeneous disease in which genetic alterations play a key role for diagnosis, prognosis, and therapy options. Fms-Like Tyrosine Kinase 3 (FLT3), Nucleophosmin 1 (NPM1), and isocitrate dehydrogenase 2 (IDH2) mutations have clinical significance by influencing the biologi...
Hero Hamad, G. Othman, R. Polus
· Iraqi Journal of Hematology · 0 citations