Alzheimer's disease (AD) is characterized by progressive cognitive decline associated with synaptic and large-scale network dysfunction. Resting-state alterations may provide functional correlates of AD pathology, but associations among intrinsic brain activity, biomarkers, and domain-specific cognition have not been s...
Soraya Alfonsín, Brenda Chino, J. de Frutos-Lucas et al.· NeuroImage: Clinical· 0 citations
The convergence of candidate genes on synaptic, neurodevelopmental and neuroinflammatory pathways, and genetically informed clustering linked to plasma p-tau217 and delayed verbal recall, supports the use of EEG-derived endophenotypes for dissecting heterogeneity in early cognitive decline and AD-related pathology.
A. Siitonen, T. Saarinen, M. Liljestrom et al.· medRxiv· 0 citations
The findings indicate that a robust four-factor cognitive structure can be identified in clinically defined MCI cohorts without prior biological selection, and the association between latent memory factors and plasma p-tau217, observed primarily in cohorts with higher biomarker burden or clearer amnestic profiles, high...
Ana S. Perez, Hugo L. Hammer, V. Andersson et al.· GeroScience· 0 citations
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