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Author

Doeun Kim

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Open access Aug 2026

Neuronal AIMP2–α-synuclein synergy drives endogenous tau and amyloid-β pathologies and neurodegeneration in a mechanistic model of Lewy body dementia

A genetically controlled in vivo model of LBD-like mixed proteinopathy recapitulating αSyn aggregation, tau hyperphosphorylation, Aβ accumulation, neurodegeneration, and behavioral decline is established and supports a mechanistic link between AIMP2-enhanced αSyn toxicity and secondary proteinopathies.

Doeun Kim, Hee-Tae Kim, Ji Hun Kim et al. · 0 citations

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