Author

Bowen Sun

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Open access Aug 2026

MFN2 alleviates mitochondrial calcium overload in sepsis-induced myopathy by modulating GRP75–VDAC1 interaction

Findings identify MFN2 as a potential regulator of MAM-mediated mitochondrial calcium homeostasis in SIM and suggest it may represent a therapeutic target for sepsis-induced skeletal muscle dysfunction.

Xuexin Li, Fei He, Yun Wang et al. · Skeletal Muscle · 0 citations