[Transcriptomic analysis of NT5E/CD73-related macrophage state remodeling in the maternal-fetal immune microenvironment of recurrent pregnancy loss].
The findings suggest that the adenosine-NT5E/CD73 axis may be a candidate pathway related to immune imbalance in RSA, but the current evidence cannot replace validation of NT5E/CD73 protein expression and spatial colocalization of CD68/CD86/CD206 in patient villous tissues.