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The mechanisms underlying cuproptosis are elucidated, myosin heavy-chain 9 (MYH9) is established as an executioner, and a treatment strategy for drug-resistant tumors is proposed.
RNA sequencing of lenvatinib-resistant HCC cell lines revealed p53 dysregulation, and sanger sequencing identified the p53 R280S missense mutation, which was shown to enhance lenvatinib resistance in vitro and in vivo.
It is found that lncRNA MKRN3-AS1 was dramatically upregulated in HCC tissues and associated with poor prognosis, and may be a potential prognostic biomarker and therapeutic target for HCC.
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