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Brain‐derived p‐tau217 may reduce these off‐target effects of Alzheimer's disease pathology, but its performance against neuropathology has not been evaluated.
Findings support the potential value of EOAD-signature atrophy as an MRI-based biomarker for individualized prognostication and clinical trial stratification and support the potential value of EOAD-signature atrophy as an MRI-based biomarker for individualized prognostication and clinical trial stratification.
It is found that women with Parkinson's disease Dementia are more vulnerable to developing Alzheimer's-like brain changes than men with Parkinson's disease Dementia and the need for sex-informed approaches to reseach in mixed neurodegenerative disease is supported.
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